The role of IP3R-SOCCs in Cr(vi)-induced cytosolic Ca2+ overload and apoptosis in L-02 hepatocytes-Reference-Cited by-同舟云学术

The role of IP3R-SOCCs in Cr(vi)-induced cytosolic Ca2+ overload and apoptosis in L-02 hepatocytes

Published:2018 Issue:3 Volume:7 Page:521-528
ISSN:2045-452X
Container-title:Toxicology Research
language:en
Short-container-title:Toxicol. Res.

Author:

Liang Qi¹²³⁴⁵,Zhang Yujing⁶⁷³⁸⁵,Zeng Ming⁶⁷³⁸⁵,Guan Lan⁶⁷³⁸⁵,Xiao Yuanyuan⁶⁷³⁸⁵,Xiao Fang⁶⁷³⁸⁵^ORCID

Affiliation:

1. Department of Radiology

2. The Third Xiangya Hospital

3. Central South University

4. Changsha 410013

5. PR China

6. Department of Health Toxicology

7. Xiangya School of Public Health

8. Changsha 410078

Abstract

Heavy metals such as hexavalent chromium [Cr(vi)] could induce Ca²⁺ overload and subsequently hepatocyte injury, and even apoptotic cell death, but the source of the increased cytosolic-free Ca²⁺ is still unclear.

Funder

National Natural Science Foundation of China

Publisher

Oxford University Press (OUP)

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

http://pubs.rsc.org/en/content/articlepdf/2018/TX/C8TX00029H

Reference25 articles.

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3. Oral exposure to hexavalent chromium through drinking water and urologic morbidity in an industrial area of Greece

4. The versatility and universality of calcium signalling

5. Calcium signalling: dynamics, homeostasis and remodelling

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