Myricetin ameliorates brain injury and neurological deficits via Nrf2 activation after experimental stroke in middle-aged rats-Reference-Cited by-同舟云学术

Myricetin ameliorates brain injury and neurological deficits via Nrf2 activation after experimental stroke in middle-aged rats

Published:2016 Issue:6 Volume:7 Page:2624-2634
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Wu Shuangchan¹²³⁴⁵,Yue Yuan¹²³⁴⁵,Peng Anlin¹²³⁴⁵,Zhang Lu¹²³⁴⁵,Xiang Jin¹²³⁴⁵,Cao Xiaoqin⁶⁷⁵⁸,Ding Hong¹²³⁴⁵,Yin Shanye⁹¹⁰¹¹¹²

Affiliation:

1. Key Laboratory of Combinatorial Biosynthesis and Drug Discovery

2. Ministry of Education

3. Wuhan University School of Pharmaceutical Sciences

4. Wuhan University

5. Wuhan

6. College of medicine

7. Jianghan University

8. P. R. China

9. Department of Cell Biology

10. Harvard Medical School

11. Boston

12. USA

Abstract

The aim of our study was to investigate the protective effects and underlying mechanisms of myricetin, a bioactive food compound, on brain injury and neurological deficits after ischemic stroke.

Publisher

Royal Society of Chemistry (RSC)

Subject

General Medicine,Food Science

Link

http://pubs.rsc.org/en/content/articlepdf/2016/FO/C6FO00419A

Reference55 articles.

1. Executive Summary: Heart Disease and Stroke Statistics—2013 Update

2. Tramiprosate protects neurons against ischemic stroke by disrupting the interaction between PSD95 and nNOS

3. Protective Effects of Myricetin on Acute Hypoxia-Induced Exercise Intolerance and Mitochondrial Impairments in Rats

4. Safety assessment and single-dose toxicokinetics of the flavouring agent myricitrin in Sprague–Dawley rats

5. Myricetin affords protection against peroxynitrite-mediated DNA damage and hydroxyl radical formation

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