GVS-12 attenuates non-alcoholic steatohepatitis by suppressing inflammatory responses via PPARγ/STAT3 signaling pathways
Author:
Affiliation:
1. Guangxi Colleges and Universities Key Laboratory of Pharmacology
2. Guilin Medical University
3. Guilin 541004
4. China
Abstract
Non-alcoholic steatohepatitis (NASH), a type of fatty liver disease, is characterized by excessive inflammation and fat accumulation in the liver.
Funder
National Natural Science Foundation of China
Natural Science Foundation of Guangxi Province
Publisher
Royal Society of Chemistry (RSC)
Subject
General Chemical Engineering,General Chemistry
Link
http://pubs.rsc.org/en/content/articlepdf/2019/RA/C8RA10178G
Reference34 articles.
1. Pathophysiology and Mechanisms of Nonalcoholic Fatty Liver Disease
2. Molecular Pathogenesis of NASH
3. From the metabolic syndrome to NAFLD or vice versa?
4. Systematic review: the epidemiology and natural history of non-alcoholic fatty liver disease and non-alcoholic steatohepatitis in adults
5. Lysyl–Transfer RNA Synthetase Induces the Maturation of Dendritic Cells through MAPK and NF-κB Pathways, Strongly Contributing to Enhanced Th1 Cell Responses
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