Copper intoxication inhibits aerobic nucleotide synthesis in Streptococcus pneumoniae
Author:
Affiliation:
1. Department of Infectious Diseases
2. St. Jude Children's Research Hospital
3. Memphis, USA
4. Department of Microbiology
5. University of Illinois Urbana-Champaign
6. Urbana, USA
Abstract
Graphical model of copper export and toxicity in S. pneumoniae.
Publisher
Oxford University Press (OUP)
Subject
Metals and Alloys,Biochemistry,Biomaterials,Biophysics,Chemistry (miscellaneous)
Link
http://pubs.rsc.org/en/content/articlepdf/2015/MT/C5MT00011D
Reference54 articles.
1. Role of the manganese efflux system mntE for signalling and pathogenesis in Streptococcus pneumoniae
2. The cop operon is required for copper homeostasis and contributes to virulence in Streptococcus pneumoniae
3. Resistance mechanisms of Mycobacterium tuberculosis against phagosomal copper overload
4. Copper at the Front Line of the Host-Pathogen Battle
5. The roles of transition metals in the physiology and pathogenesis of Streptococcus pneumoniae
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