S-Methyl-l-cysteine targeting MsrA attenuates Ang II-induced oxidative stress and atrial remodeling via the p38 MAPK signaling pathway

Author:

Xu Beibei12,Xu Yinli1,Ren Wenpu13,Meng Shan14,Hong Tao56,Cao Zijun3,Xiao Xiong17,Guo Xiaodong5,Yu Liming1ORCID,Zhao Jikai1ORCID,Wang Huishan1ORCID

Affiliation:

1. State Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theatre Command, Shenyang 110016, China

2. Postgraduate College, Shenyang Pharmaceutical University, Shenyang 110016, China

3. Postgraduate College, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China

4. Postgraduate College, Jinzhou Medical University, Jinzhou 121001, China

5. Postgraduate College, Dalian Medical University, Dalian 116044, China

6. Pediatric Surgery Ward, Fuwai Hospital Chinese Academy of Medical Sciences, Shenzhen 518052, China

7. Postgraduate College, China Medical University, Shenyang 110122, China

Abstract

SMLC protects against atrial remodeling of AF by inhibiting oxidative stress through the mediation of the MsrA/p38 MAPK signaling pathway

Funder

National Natural Science Foundation of China

Natural Science Foundation of Liaoning Province

Publisher

Royal Society of Chemistry (RSC)

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