Ginsenoside Rb1 prevents hypoxia-reoxygenation-induced apoptosis in H9c2 cardiomyocytes via an estrogen receptor-dependent crosstalk among the Akt, JNK, and ERK 1/2 pathways using a label-free quantitative proteomics analysis-Reference-Cited by-同舟云学术

Ginsenoside Rb1 prevents hypoxia-reoxygenation-induced apoptosis in H9c2 cardiomyocytes via an estrogen receptor-dependent crosstalk among the Akt, JNK, and ERK 1/2 pathways using a label-free quantitative proteomics analysis

Published:2015 Issue:33 Volume:5 Page:26346-26363
ISSN:2046-2069
Container-title:RSC Advances
language:en
Short-container-title:RSC Adv.

Author:

Ai Qidi¹²³⁴⁵,Sun Guibo¹²³⁴⁵,Luo Yun¹²³⁴⁵,Dong Xi⁶⁷⁸,Hu Ruifeng¹²³⁴⁵,Meng Xiangbao¹²³⁴⁵,Sun Xiaobo¹²³⁴⁵

Affiliation:

1. Key Laboratory of Bioactive Substances and Resources Utilization of Chinese Herbal Medicine

2. Ministry of Education

3. Institute of Medicinal Plant Development

4. Chinese Academy of Medical Sciences & Peking Union Medical College

5. Beijing

6. Wenzhou Medical University

7. Wenzhou

8. P. R. China

Abstract

Rb1 prevents H/R-induced apoptosis of H9c2 cells via an estrogen receptor-dependent crosstalk among the Akt, JNK, and ERK 1/2 pathways.

Publisher

Royal Society of Chemistry (RSC)

Subject

General Chemical Engineering,General Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2015/RA/C5RA02432C

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4. LipoxinA4 attenuates myocardial ischemia reperfusion injury via a mechanism related to downregulation of GRP-78 and caspase-12 in rats

5. Notoginsenoside R1-mediated neuroprotection involves estrogen receptor-dependent crosstalk between Akt and ERK1/2 pathways: A novel mechanism of Nrf2/ARE signaling activation

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