MiR-146a affects the alteration in myeloid differentiation induced by hydroquinone in human CD34+hematopoietic progenitor cells and HL-60 cells
Author:
Affiliation:
1. Guangzhou Key Laboratory of Environmental Pollution and Health Risk Assessment
2. School of Public Health
3. Sun Yat-sen University
4. Guangzhou
5. China
6. Department of Obstetrics and Gynecology
7. Guangdong General Hospital
Abstract
The MiR-146a/TRAF6/NF-κB axis is important for the regulation of hematopoiesis and the immune system.
Publisher
Oxford University Press (OUP)
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://pubs.rsc.org/en/content/articlepdf/2016/TX/C5TX00419E
Reference42 articles.
1. Current understanding of the mechanism of benzene-induced leukemia in humans: implications for risk assessment
2. Effects of benzene metabolite treatment on granulocytic differentiation and DNA adduct formation in HL-60 cells
3. Hydroquinone, a benzene metabolite, increases the level of aneusomy of chromosomes 7 and 8 in human CD34-positive blood progenitor cells
4. Hydroquinone, a Bioreactive Metabolite of Benzene, Inhibits Apoptosis in Myeloblasts
5. Phenolic metabolites of benzene inhibited the erythroid differentiation of K562 cells
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