GLY67ARG substitution in RSPO4 disrupts the WNT signaling pathway due to an abnormal binding pattern with LGRs leading to anonychia-Reference-Cited by-同舟云学术

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GLY67ARG substitution in RSPO4 disrupts the WNT signaling pathway due to an abnormal binding pattern with LGRs leading to anonychia

Published:2017 Issue:28 Volume:7 Page:17357-17366
ISSN:2046-2069
Container-title:RSC Advances
language:en
Short-container-title:RSC Adv.

Author:

Raza Syed Irfan¹²³⁴⁵,Navid Abdul Khaliq⁶⁷⁴⁵,Noor Zainab⁸³⁴⁵,Shah Khadim¹²³⁴⁵,Dar Nasser Rashid⁹¹⁰¹¹⁵,Ahmad Wasim¹²³⁴⁵,Rashid Sajid⁸³⁴⁵^ORCID

Affiliation:

1. Department of Biochemistry

2. Faculty of Biological Sciences

3. Quaid-i-Azam University

4. Islamabad

5. Pakistan

6. Islamic Medical College

7. Riffah University

8. National Centre for Bioinformatics

9. Department of Dermatology

10. Combined Military Hospital

11. Rawalpindi

Abstract

R-Spondins regulate the WNT/β-catenin signaling pathway by interacting with leucine rich-repeat containing G-protein coupled receptors (LGR4–6).

Publisher

Royal Society of Chemistry (RSC)

Subject

General Chemical Engineering,General Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2017/RA/C7RA00762K

Reference59 articles.

1. R-Spondin2 Is a Secreted Activator of Wnt/β-Catenin Signaling and Is Required for Xenopus Myogenesis

2. R-Spondin Proteins: A Novel Link to β-catenin Activation

3. Mouse Cristin/R-spondin Family Proteins Are Novel Ligands for the Frizzled 8 and LRP6 Receptors and Activate β-Catenin-dependent Gene Expression

4. R-Spondin1 regulates Wnt signaling by inhibiting internalization of LRP6

5. R-spondin1 Is a High Affinity Ligand for LRP6 and Induces LRP6 Phosphorylation and β-Catenin Signaling

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