Copper-induced cell death and the protective role of glutathione: the implication of impaired protein folding rather than oxidative stress-Reference-Cited by-同舟云学术

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Copper-induced cell death and the protective role of glutathione: the implication of impaired protein folding rather than oxidative stress

Published:2018 Issue:12 Volume:10 Page:1743-1754
ISSN:1756-5901
Container-title:Metallomics
language:en
Short-container-title:Metallomics

Author:

Saporito-Magriñá Christian Martín¹²³⁴⁵^ORCID,Musacco-Sebio Rosario Natalia¹²³⁴⁵^ORCID,Andrieux Geoffroy⁶⁷⁸⁹¹⁰^ORCID,Kook Lucas⁶⁷⁸⁹¹⁰,Orrego Manuel Tomás¹²¹¹¹²⁵^ORCID,Tuttolomondo María Victoria¹²³¹³¹⁴^ORCID,Desimone Martín Federico¹²³¹³¹⁴^ORCID,Boerries Melanie⁶⁷⁸⁹¹⁰^ORCID,Borner Christoph⁶⁷⁸⁹¹⁰^ORCID,Repetto Marisa Gabriela¹²³⁴⁵^ORCID

Affiliation:

1. Universidad de Buenos Aires

2. Facultad de Farmacia y Bioquímica

3. Departamento de Química Analítica y Fisicoquímica

4. Cátedra de Química General e Inorgánica

5. Buenos Aires

6. Institute of Molecular Medicine and Cell Research

7. Faculty of Medicine

8. Albert Ludwigs University of Freiburg

9. D-79104 Freiburg

10. Germany

11. Departamento de Fisicomatemática

12. Cátedra de Física

13. Cátedra de Química Analítica Instrumental

14. CONICET-UBA

Abstract

Intracellular mechanisms of copper cytotoxicity.

Funder

Universidad de Buenos Aires

Deutsche Forschungsgemeinschaft

Consejo Nacional de Investigaciones Científicas y Técnicas

Deutscher Akademischer Austauschdienst

Bundesministerium für Bildung und Forschung

Publisher

Oxford University Press (OUP)

Subject

Metals and Alloys,Biochemistry,Biomaterials,Biophysics,Chemistry (miscellaneous)

Link

http://pubs.rsc.org/en/content/articlepdf/2018/MT/C8MT00182K

Reference57 articles.

1. Structure of the Ctr1 copper trans‘PORE’ter reveals novel architecture

2. Cloning and characterization of a mammalian proton-coupled metal-ion transporter

3. Function and Regulation of Human Copper-Transporting ATPases

4. Affinity gradients drive copper to cellular destinations

5. Diagnosis of Wilson's disease: an experience over three decades

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