Destructive fibrotic teamwork: how both microenvironment stiffness and profibrotic interleukin 13 impair alveolar macrophage phenotype and function-Reference-Cited by-同舟云学术

Destructive fibrotic teamwork: how both microenvironment stiffness and profibrotic interleukin 13 impair alveolar macrophage phenotype and function

Published:2022 Issue:19 Volume:10 Page:5689-5706
ISSN:2047-4830
Container-title:Biomaterials Science
language:en
Short-container-title:Biomater. Sci.

Author:

Bomb Kartik¹^ORCID,Pradhan Lina¹,Zhang Qi¹,Jarai Bader M.¹,Bhattacharjee Arnab²,Burris David L.²,Kloxin April M.¹³^ORCID,Fromen Catherine A.¹^ORCID

Affiliation:

1. Chemical and Biomolecular Engineering, University of Delaware, Newark, DE, USA

2. Mechanical Engineering, University of Delaware, Newark, DE, USA

3. Material Science and Engineering, University of Delaware, Newark, DE, USA

Abstract

Alternative activation (M2 phenotype) of macrophages and reduced phagocytosis is regulated synergistically by increased substrate stiffness and presence of profibrotic soluble cues.

Funder

NIH Office of the Director

Division of Materials Research

National Institute of General Medical Sciences

Publisher

Royal Society of Chemistry (RSC)

Subject

General Materials Science,Biomedical Engineering

Link

http://pubs.rsc.org/en/content/articlepdf/2022/BM/D2BM00828A

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