Modulation of JNK-1/ β-catenin signaling byLactobacillus casei, inulin and their combination in 1,2-dimethylhydrazine-induced colon cancer in mice-Reference-Cited by-同舟云学术

Modulation of JNK-1/ β-catenin signaling byLactobacillus casei, inulin and their combination in 1,2-dimethylhydrazine-induced colon cancer in mice

Published:2019 Issue:50 Volume:9 Page:29368-29383
ISSN:2046-2069
Container-title:RSC Advances
language:en
Short-container-title:RSC Adv.

Author:

Ali Mohammed S.¹²³⁴⁵,Hussein Rasha M.¹²³⁴⁵^ORCID,Gaber Yasser⁶⁷⁸⁹¹⁰^ORCID,Hammam Olfat A.¹¹¹²¹³⁵,Kandeil Mohamed A.¹¹⁴³⁵

Affiliation:

1. Department of Biochemistry

2. Faculty of Pharmacy

3. Beni-Suef University

4. Beni-Suef

5. Egypt

6. Department of Pharmaceutics and Pharmaceutical Technology

7. College of Pharmacy

8. Mutah University

9. Al-Karak

10. Jordan

11. Pathology Department

12. Theodor Bilharz Research Institute

13. 12411 Giza

14. Faculty of Veterinary Medicine

Abstract

Thelactobacillus caseiand inulin modulate the expression of JNK-1, GSK3β and β-catenin proteins and enrich the beneficial bacteria to protect from colon cancer in mice.

Publisher

Royal Society of Chemistry (RSC)

Subject

General Chemical Engineering,General Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2019/RA/C9RA04388H

Reference82 articles.

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2. Silibinin ameliorates oxidative stress induced aberrant crypt foci and lipid peroxidation in 1, 2 dimethylhydrazine induced rat colon cancer

3. Microbiota modification by probiotic supplementation reduces colitis associated colon cancer in mice

4. Phytic acid improves intestinal mucosal barrier damage and reduces serum levels of proinflammatory cytokines in a 1,2-dimethylhydrazine-induced rat colorectal cancer model

5. Parabacteroides distasonis attenuates toll‐like receptor 4 signaling and Akt activation and blocks colon tumor formation in high‐fat diet‐fed azoxymethane‐treated mice

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