SLL-1A-16 suppresses proliferation and induces autophagy in non-small-cell lung cancer cells <i>via</i> the AKT/mTOR signaling pathway-Reference-Cited by-同舟云学术

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SLL-1A-16 suppresses proliferation and induces autophagy in non-small-cell lung cancer cells via the AKT/mTOR signaling pathway

Published:2024 Issue: Volume: Page:
ISSN:2632-8682
Container-title:RSC Medicinal Chemistry
language:en
Short-container-title:RSC Med. Chem.

Author:

Luo Xiaoqin¹,Wang Jin¹,Wang Ruichang¹,Lian Jiabing¹,Guo Mengnan¹,Zhou Hongrui¹,Zhang Mengxue¹,Yang Zhe¹^ORCID,Li Xiaolong²,He Xianran³,Bi Xiuli¹⁴^ORCID

Affiliation:

1. College of Life Science, Liaoning University, 66 Chongshan Road, Shenyang 110036, China

2. Shenzhen Fushan Biological Technology Co., Ltd, Kexing Science Park A1 1005, Nanshan Zone, Shenzhen 518057, China

3. Institute for Interdisciplinary Research, Jianghan University, Wuhan Economic and Technological Development Zone, Wuhan 430056, China

4. Key Laboratory of Chronic Disease Occurrence and Nutrition Intervention, Liaoning University, 66 Chongshan Road, Shenyang 110036, China

Abstract

Schematic of proposed mechanism for the treatment of the SLL-1A-16 inhibiting the proliferation in NSCLC.

Funder

Liaoning Revitalization Talents Program

Publisher

Royal Society of Chemistry (RSC)

Link

http://pubs.rsc.org/en/content/articlepdf/2024/MD/D4MD00405A

Reference41 articles.

1. Lung Cancer 2020

2. Current and Future Development in Lung Cancer Diagnosis

3. Update 2020: Management of Non-Small Cell Lung Cancer

4. The Role of mTOR Signaling as a Therapeutic Target in Cancer

5. Regulation and metabolic functions of mTORC1 and mTORC2

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