Se-methylselenocysteine ameliorates mitochondrial function by targeting both mitophagy and autophagy in the mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Se-methylselenocysteine ameliorates mitochondrial function by targeting both mitophagy and autophagy in the mouse model of Alzheimer's disease

Published:2024 Issue:8 Volume:15 Page:4310-4322
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Xie Yongli¹,Ke Xiaoshan¹,Ye Zhencong¹,Li Xuexia¹,Chen Zetao¹^ORCID,Liu Jiantao¹^ORCID,Wu Ziyi¹,Liu Qiong¹²,Du Xiubo¹

Affiliation:

1. Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen 518055, China

2. Shenzhen-Hong Kong Institute of Brain Science—Shenzhen Fundamental Research Institutions, Shenzhen 518055, China

Abstract

This study aimed to investigate the therapeutic effects and molecular mechanisms of SMC on mitochondrial function in 3 × Tg-AD mice.

Publisher

Royal Society of Chemistry (RSC)

Link

http://pubs.rsc.org/en/content/articlepdf/2024/FO/D4FO00520A

Reference81 articles.

1. Alzheimer disease

2. Alzheimer’s Disease: From Firing Instability to Homeostasis Network Collapse

3. Inflammation in Alzheimer Disease--A Brief Review of the Basic Science and Clinical Literature

4. Reversal of autophagy dysfunction in the TgCRND8 mouse model of Alzheimer's disease ameliorates amyloid pathologies and memory deficits

5. Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease

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