Hesperetin attenuates sepsis-induced intestinal barrier injury by regulating neutrophil extracellular trap formation <i>via</i> the ROS/autophagy signaling pathway-Reference-Cited by-同舟云学术

Hesperetin attenuates sepsis-induced intestinal barrier injury by regulating neutrophil extracellular trap formation via the ROS/autophagy signaling pathway

Published:2023 Issue:9 Volume:14 Page:4213-4227
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Chen Fang¹^ORCID,Chu Chengnan²,Wang Xinyu²,Yang Chao²,Deng Yunxuan²,Duan Zehua²,Wang Kai²,Liu Baochen²,Ji Wu²,Ding Weiwei¹²^ORCID

Affiliation:

1. Jinling Hospital, School of Medicine, Southeast University, and State Key Laboratory of Trauma, Burn and Combined Injury, P. R. China

2. Division of Trauma and Surgical Intensive Care Unit, Research Institute of General Surgery, Jinling Hospital, Affiliated Hospital of Medical School, Nanjing University, Nanjing 210002, Jiangsu Province, P. R. China

Abstract

Hesperetin (HES), one of the major flavonoids that has various biological activities, such as anti-inflammatory and antioxidant activities, may preserve the intestinal barrier during sepsis.

Funder

National Natural Science Foundation of China

State Key Laboratory of Trauma, Burns and Combined Injury

National Key Clinical Specialty Discipline Construction Program of China

Publisher

Royal Society of Chemistry (RSC)

Subject

General Medicine,Food Science

Link

http://pubs.rsc.org/en/content/articlepdf/2023/FO/D2FO02707K

Reference67 articles.

1. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)