Knockdown of FOXO6 inhibits cell proliferation and ECM accumulation in glomerular mesangial cells cultured under high glucose condition
Author:
Affiliation:
1. Department of Nephrology
2. Huaihe Hospital of Henan University
3. Kaifeng 475000
4. P. R. China
5. Department of Endocrinology
Abstract
Forkhead box O 6 (FOXO6), a FOX transcription factor, has been found to be involved in diabetes mellitus and related complications.
Publisher
Royal Society of Chemistry (RSC)
Subject
General Chemical Engineering,General Chemistry
Link
http://pubs.rsc.org/en/content/articlepdf/2019/RA/C8RA10547B
Reference26 articles.
1. Mechanisms of Diabetic Complications
2. Clinical therapeutic strategies for early stage of diabetic kidney disease
3. Diabetic Nephropathy: Diagnosis, Prevention, and Treatment
4. A Glimpse of Various Pathogenetic Mechanisms of Diabetic Nephropathy
5. Pathogenetic Mechanisms of Diabetic Nephropathy
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1. Overexpression of FTO inhibits excessive proliferation and promotes the apoptosis of human glomerular mesangial cells by alleviating FOXO6 m6A modification via YTHDF3-dependent mechanisms;Frontiers in Pharmacology;2023-09-26
2. Fibrosis: Sirtuins at the checkpoints of myofibroblast differentiation and profibrotic activity;Wound Repair and Regeneration;2021-06-10
3. FOXP1 inhibits high glucose-induced ECM accumulation and oxidative stress in mesangial cells;Chemico-Biological Interactions;2019-11
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