Silencing of USP22 suppresses high glucose-induced apoptosis, ROS production and inflammation in podocytes
Author:
Affiliation:
1. Department of Endocrinology
2. Changhai Hospital
3. The Second Military Medical University
4. Shanghai 200433
5. China
Abstract
Silencing of USP22 suppresses high glucose-induced podocyte injury.
Publisher
Royal Society of Chemistry (RSC)
Subject
Molecular Biology,Biotechnology
Link
http://pubs.rsc.org/en/content/articlepdf/2016/MB/C5MB00722D
Reference29 articles.
1. The Podocyte: A Major Player in the Development of Diabetic Nephropathy?
2. Glucose-Induced Reactive Oxygen Species Cause Apoptosis of Podocytes and Podocyte Depletion at the Onset of Diabetic Nephropathy
3. Dysregulated Nephrin in Diabetic Nephropathy of Type 2 Diabetes: A Cross Sectional Study
4. Urinary excretion of podocytes in patients with diabetic nephropathy
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