Integrating qualitative and quantitative assessments of Yougui pill, an effective traditional Chinese medicine, by HPLC-LTQ-Orbitrap-MSn and UPLC-QqQ-MS/MS-Reference-Cited by-同舟云学术

Integrating qualitative and quantitative assessments of Yougui pill, an effective traditional Chinese medicine, by HPLC-LTQ-Orbitrap-MSn and UPLC-QqQ-MS/MS

Published:2017 Issue:23 Volume:9 Page:3485-3496
ISSN:1759-9660
Container-title:Analytical Methods
language:en
Short-container-title:Anal. Methods

Author:

Liu Haolong¹²³⁴⁵,Qiu Feng¹²³⁴⁵,Bian Baolin⁶⁷⁸⁵,Yang Xinwei¹²³⁴⁵,Zou Haiyan¹²³⁴⁵,Zhao Hui¹²³⁴⁵,Chen Zhenzhen¹²³⁴⁵,Wang Qingsong⁹³⁴⁵,Zhao Haiyu⁶⁷⁸⁵,Wang Lei¹²³⁴⁵^ORCID

Affiliation:

1. School of Traditional Chinese Medicine

2. Beijing Key Lab of TCM Collateral Disease Theory Research

3. Capital Medical University

4. Beijing 100069

5. China

6. Institute of Chinese Materia Medica

7. China Academy of Chinese Medical Sciences

8. Beijing 100700

9. Basic Medical Experimental Teaching Center

Abstract

Yougui pill (YGP) is a well-known Chinese medicine formula with definite clinical effects on kidney-yang deficiencies such as infertility.

Funder

Natural Science Foundation of Beijing Municipality

Publisher

Royal Society of Chemistry (RSC)

Subject

General Engineering,General Chemical Engineering,Analytical Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2017/AY/C7AY00259A

Reference44 articles.

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2. Chinese Pharmacopeia Commission , Pharmacopeia of the People's Republic of China (Version 2015), China Medical Science Press, Beijing, 2015, pp. 767–768

3. Zuo-Gui and You-Gui pills, two traditional Chinese herbal formulas, downregulated the expression of NogoA, NgR, and RhoA in rats with experimental autoimmune encephalomyelitis

4. Effects of Zuogui Pill (左归丸) and Yougui Pill (右归丸) on the expression of brain-derived neurotrophic factor and cyclic adenosine monophosphate/protein kinase a signaling transduction pathways of axonal regeneration in model rats with experimental autoimmune encephalomyelitis

5. You-Gui pills promote nerve regeneration by regulating netrin1, DCC and Rho family GTPases RhoA, Racl, Cdc42 in C57BL/6 mice with experimental autoimmune encephalomyelitis

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