Chimaeric mice deficient in dystroglycans develop muscular dystrophy and have disrupted myoneural synapses
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng1199_338.pdf
Reference31 articles.
1. Koenig, M. et al. Complete cloning of the Duchenne muscular dystrophy (DMD ) cDNA and preliminary genomic organization of the DMD gene in mouse and affected individuals. Cell 50, 509– 517 (1987).
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3. Gee, S.H., Montanaro, F., Lindenbaum, M.H. & Carbonetto, S. Dystroglycan-α, a dystrophin-associated glycoprotein, is a functional agrin receptor. Cell 77, 675– 686 (1994).
4. Campanelli, J.T., Roberds, S.L., Campbell, K.P. & Scheller, R.H. A role for dystrophin associated glycoproteins and utrophin in agrin-induced AChR clustering. Cell 77, 663– 674 (1994).
5. Williamson R.A. et al. Dystroglycan is essential for early embryonic development—disruption of Reichert's membrane in Dag1-null mice. Hum. Mol. Genet. 6, 831–841 ( 1997).
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