Autistic-like social behaviour in Shank2-mutant mice improved by restoring NMDA receptor function-Reference-Cited by-同舟云学术

Autistic-like social behaviour in Shank2-mutant mice improved by restoring NMDA receptor function

Published:2012-06 Issue:7402 Volume:486 Page:261-265
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Won Hyejung,Lee Hye-Ryeon,Gee Heon Yung,Mah Won,Kim Jae-Ick,Lee Jiseok,Ha Seungmin,Chung Changuk,Jung Eun Suk,Cho Yi Sul,Park Sae-Geun,Lee Jung-Soo,Lee Kyungmin,Kim Daesoo,Bae Yong Chul,Kaang Bong-Kiun,Lee Min Goo,Kim Eunjoon

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature11208.pdf

Reference30 articles.

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3. Boeckers, T. M., Bockmann, J., Kreutz, M. R. & Gundelfinger, E. D. ProSAP/Shank proteins—a family of higher order organizing molecules of the postsynaptic density with an emerging role in human neurological disease. J. Neurochem. 81, 903–910 (2002)

4. Ehlers, M. D. Synapse structure: glutamate receptors connected by the shanks. Curr. Biol. 9, R848–R850 (1999)

5. Berkel, S. et al. Mutations in the SHANK2 synaptic scaffolding gene in autism spectrum disorder and mental retardation. Nature Genet. 42, 489–491 (2010)

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