Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes-Reference-Cited by-同舟云学术

Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes

Published:2010-09-12 Issue:10 Volume:11 Page:897-904
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Masters Seth L,Dunne Aisling,Subramanian Shoba L,Hull Rebecca L,Tannahill Gillian M,Sharp Fiona A,Becker Christine,Franchi Luigi,Yoshihara Eiji,Chen Zhe,Mullooly Niamh,Mielke Lisa A,Harris James,Coll Rebecca C,Mills Kingston H G,Mok K Hun,Newsholme Philip,Nuñez Gabriel,Yodoi Junji,Kahn Steven E,Lavelle Ed C,O'Neill Luke A J

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.1935.pdf

Reference53 articles.

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2. Njajou, O.T. et al. Association between oxidized LDL, obesity and type 2 diabetes in a population-based cohort, the Health, Aging and Body Composition Study. Diabetes Metab. Res. Rev. 25, 733–739 (2009).

3. Bendtzen, K. et al. Cytotoxicity of human pI 7 interleukin-1 for pancreatic islets of Langerhans. Science 232, 1545–1547 (1986).

4. Spranger, J. et al. Inflammatory cytokines and the risk to develop type 2 diabetes: results of the prospective population-based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study. Diabetes 52, 812–817 (2003).

5. Larsen, C.M. et al. Interleukin-1-receptor antagonist in type 2 diabetes mellitus. N. Engl. J. Med. 356, 1517–1526 (2007).

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