Inflammation-activated CXCL16 pathway contributes to tubulointerstitial injury in mouse diabetic nephropathy-Reference-Cited by-同舟云学术

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Inflammation-activated CXCL16 pathway contributes to tubulointerstitial injury in mouse diabetic nephropathy

Published:2018-04-05 Issue:6 Volume:39 Page:1022-1033
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Hu Ze-bo,Ma Kun-ling,Zhang Yang,Wang Gui-hua,Liu Liang,Lu Jian,Chen Pei-pei,Lu Chen-chen,Liu Bi-cheng

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

http://www.nature.com/articles/aps2017177.pdf

Reference41 articles.

1. Kitada M, Ogura Y, Monno I, Koya D . Regulating autophagy as a therapeutic target for diabetic nephropathy. Curr Diab Rep 2017; 17: 53.

2. Mauer M, Caramori ML, Fioretto P, Najafian B . Glomerular structural-functional relationship models of diabetic nephropathy are robust in type 1 diabetic patients. Nephrol Dial Transplant 2015; 30: 918–23.

3. Cao Z, Cooper ME . Pathogenesis of diabetic nephropathy. J Diabetes Investig 2011; 2: 243–7.

4. Yacoub R, Campbell KN . Inhibition of RAS in diabetic nephropathy. Int J Nephrol Renovasc Dis 2015; 8: 29–40.

5. Ferrao F M, Lara L S, Lowe J . Renin-angiotensin system in the kidney: What is new? World J Nephrol 2014; 3: 64–76.

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