Alzheimer’s disease modification mediated by bone marrow-derived macrophages via a TREM2-independent pathway in mouse model of amyloidosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pulmonary and Respiratory Medicine,Pediatrics, Perinatology, and Child Health
Link
https://www.nature.com/articles/s43587-021-00149-w.pdf
Reference80 articles.
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3. Lesne, S. et al. A specific amyloid-beta protein assembly in the brain impairs memory. Nature 440, 352–357 (2006).
4. Bernstein, S. L. et al. Amyloid-beta protein oligomerization and the importance of tetramers and dodecamers in the aetiology of Alzheimer’s disease. Nat. Chem. 1, 326–331 (2009).
5. Shea, D. et al. alpha-Sheet secondary structure in amyloid beta-peptide drives aggregation and toxicity in Alzheimer’s disease. Proc. Natl Acad. Sci. USA 116, 8895–8900 (2019).
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