Spurious intragenic transcription is a feature of mammalian cellular senescence and tissue aging-Reference-Cited by-同舟云学术

Spurious intragenic transcription is a feature of mammalian cellular senescence and tissue aging

Published:2023-03-30 Issue:4 Volume:3 Page:402-417
ISSN:2662-8465
Container-title:Nature Aging
language:en
Short-container-title:Nat Aging

Author:

Sen Payel^ORCID,Donahue Greg,Li Catherine^ORCID,Egervari Gabor,Yang Na,Lan Yemin,Robertson Neil^ORCID,Shah Parisha P.,Kerkhoven Erik,Schultz David C.^ORCID,Adams Peter D.^ORCID,Berger Shelley L.^ORCID

Funder

U.S. Department of Health & Human Services | National Institutes of Health

American Federation for Aging Research

American Heart Association

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Geriatrics and Gerontology,Aging

Link

https://www.nature.com/articles/s43587-023-00384-3.pdf

Reference69 articles.

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3. Sen, P. et al. H3K36 methylation promotes longevity by enhancing transcriptional fidelity. Genes Dev. 29, 1362–1376 (2015).

4. Pu, M. et al. Trimethylation of Lys36 on H3 restricts gene expression change during aging and impacts life span. Genes Dev. 29, 718–731 (2015).

5. Neri, F. et al. Intragenic DNA methylation prevents spurious transcription initiation. Nature 543, 72–77 (2017).

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