Hic-5 Promotes the Hypertrophic Scar Myofibroblast Phenotype by Regulating the TGF-β1 Autocrine Loop
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference19 articles.
1. Role of transforming growth factor β in human disease;Blobe;N Engl J Med,2000
2. A TGF-β1-dependent autocrine loop regulates the structure of focal adhesions in hypertrophic scar fibroblasts;Dabiri;J Invest Dermatol,2006
3. TGF-β1 slows the growth of pathogenic myofibroblasts through a mechanism requiring the focal adhesion protein, Hic-5;Dabiri;J Invest Dermatol,2008
4. Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts;Desmoulière;J Cell Biol,1993
5. Morphological and immunochemical differences between keloid and hypertrophic scar;Ehrlich;Am J Pathol,1994
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