Reemergence of the maxi K+ as a K+ secretory channel
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference12 articles.
1. Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet;Bailey;Kidney Int,2006
2. Single channel recordings of calcium-activated potassium channels in the apical membrane of rabbit cortical collecting tubules;Hunter;Proc Natl Acad Sci,1984
3. Low-conductance K channels in apical membrane of rat cortical collecting tubule;Frindt;Am J Physiol,1989
4. Flow-dependent activation of maxi K+ channels in apical membrane of rabbit connecting tubule;Taniguchi;J Membr Biol,1998
5. Flow-dependent K+ secretion in the cortical collecting duct is mediated by a maxi-K channel;Woda;Am J Physiol Renal Physiol,2001
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2. WNK1 Activates Large-Conductance Ca2+-Activated K+ Channels through Modulation of ERK1/2 Signaling;Journal of the American Society of Nephrology;2014-08-21
3. WNK4 kinase inhibits Maxi K channel activity by a kinase-dependent mechanism;American Journal of Physiology-Renal Physiology;2011-08
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