From bench to bedside: Diagnosis of Gitelman's syndrome – defect of sodium-chloride cotransporter in renal tissue
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference20 articles.
1. Possible discriminations of Gitelman's syndrome from Bartter's syndrome by renal clearance study: report of two cases;Tsukamoto;Am J Kidney Dis,1995
2. Abnormal reabsorption of Na+/Cl− by the thiazide-inhibitable transporter of the distal convoluted tubule in Gitelman's syndrome;Colussi;Am J Nephrol,1997
3. Human cortical distal nephron: distribution of electrolyte and water transport pathways;Biner;J Am Soc Nephrol,2002
4. Vasopressin increases Na–K–2Cl cotransporter expression in thick ascending limb of Henle's loop;Kim;Am J Physiol,1999
5. A new familial disorder characterized by hypokalemia and hypomagnesemia;Gitelman;Trans Assoc Am Physicians,1966
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1. Genotypic variability in patients with clinical diagnosis of Bartter syndrome type 3;Scientific Reports;2023-08-03
2. Pathophysiology, Evaluation, and Treatment of Hypokalemia;Nephrology Self-Assessment Program;2022-01
3. Novel mutations of the SLC12A3 gene in patients with Gitelman syndrome;Scandinavian Journal of Clinical and Laboratory Investigation;2021-10-17
4. Urinary Extracellular Vesicles for Renal Tubular Transporters Expression in Patients With Gitelman Syndrome;Frontiers in Medicine;2021-06-09
5. Long-term Clinical Course after Living Kidney Donation by a Patient with Gitelman Syndrome Harboring a Compound Heterozygous Mutation of the SLC12A3 Gene;Internal Medicine;2021-05-15
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