Hepatitis-C-virus-like internal ribosome entry sites displace eIF3 to gain access to the 40S subunit-Reference-Cited by-同舟云学术

Hepatitis-C-virus-like internal ribosome entry sites displace eIF3 to gain access to the 40S subunit

Published:2013-11 Issue:7477 Volume:503 Page:539-543
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Hashem Yaser,des Georges Amedee,Dhote Vidya,Langlois Robert,Liao Hstau Y.,Grassucci Robert A.,Pestova Tatyana V.,Hellen Christopher U. T.,Frank Joachim

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature12658.pdf

Reference55 articles.

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2. Pestova, T. V., Shatsky, I. N., Fletcher, S. P., Jackson, R. J. & Hellen, C. U. A prokaryotic-like mode of cytoplasmic eukaryotic ribosome binding to the initiation codon during internal translation initiation of hepatitis C and classical swine fever virus RNAs. Genes Dev. 12, 67–83 (1998)

3. Kolupaeva, V. G., Pestova, T. V. & Hellen, C. U. Ribosomal binding to the internal ribosomal entry site of classical swine fever virus. RNA 6, 1791–1807 (2000)

4. Kolupaeva, V. G., Pestova, T. V. & Hellen, C. U. An enzymatic footprinting analysis of the interaction of 40S ribosomal subunits with the internal ribosomal entry site of hepatitis C virus. J. Virol. 74, 6242–6250 (2000)

5. Kieft, J. S., Zhou, K., Jubin, R. & Doudna, J. A. Mechanism of ribosome recruitment by hepatitis C IRES RNA. RNA 7, 194–206 (2001)

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