A stable transcription factor complex nucleated by oligomeric AML1–ETO controls leukaemogenesis-Reference-Cited by-同舟云学术

A stable transcription factor complex nucleated by oligomeric AML1–ETO controls leukaemogenesis

Published:2013-06-30 Issue:7460 Volume:500 Page:93-97
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Sun Xiao-Jian,Wang Zhanxin,Wang Lan,Jiang Yanwen,Kost Nils,Soong T. David,Chen Wei-Yi,Tang Zhanyun,Nakadai Tomoyoshi,Elemento Olivier,Fischle Wolfgang,Melnick Ari,Patel Dinshaw J.,Nimer Stephen D.,Roeder Robert G.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature12287.pdf

Reference37 articles.

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2. Peterson, L. F. & Zhang, D. E. The 8;21 translocation in leukemogenesis. Oncogene 23, 4255–4262 (2004)

3. Minucci, S. et al. Oligomerization of RAR and AML1 transcription factors as a novel mechanism of oncogenic activation. Mol. Cell 5, 811–820 (2000)

4. Liu, Y. et al. The tetramer structure of the Nervy homology two domain, NHR2, is critical for AML1/ETO’s activity. Cancer Cell 9, 249–260 (2006)

5. Kwok, C., Zeisig, B. B., Qiu, J., Dong, S. & So, C. W. Transforming activity of AML1-ETO is independent of CBFβ and ETO interaction but requires formation of homo-oligomeric complexes. Proc. Natl Acad. Sci. USA 106, 2853–2858 (2009)

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