Deletion of murine Arv1 results in a lean phenotype with increased energy expenditure-Reference-Cited by-同舟云学术

Deletion of murine Arv1 results in a lean phenotype with increased energy expenditure

Published:2015-10 Issue:10 Volume:5 Page:e181-e181
ISSN:2044-4052
Container-title:Nutrition & Diabetes
language:en
Short-container-title:Nutr & Diabetes

Author:

Lagor W R,Tong F,Jarrett K E,Lin W,Conlon D M,Smith M,Wang M Y,Yenilmez B O,McCoy M G,Fields D W,O'Neill S M,Gupta R,Kumaravel A,Redon V,Ahima R S,Sturley S L,Billheimer J T,Rader D J

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://www.nature.com/articles/nutd201532.pdf

Reference28 articles.

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2. Tinkelenberg AH, Liu Y, Alcantara F, Khan S, Guo Z, Bard M et al. Mutations in yeast ARV1 alter intracellular sterol distribution and are complemented by human ARV1. J Biol Chem 2000; 275: 40667–40670.

3. Villasmil ML, Ansbach A, Nickels JT Jr . The putative lipid transporter, Arv1, is required for activating pheromone-induced MAP kinase signaling in Saccharomyces cerevisiae. Genetics 2011; 187: 455–465.

4. Kajiwara K, Watanabe R, Pichler H, Ihara K, Murakami S, Riezman H et al. Yeast ARV1 is required for efficient delivery of an early GPI intermediate to the first mannosyltransferase during GPI assembly and controls lipid flow from the endoplasmic reticulum. Mol Biol Cell 2008; 19: 2069–2082.

5. Shechtman CF, Henneberry AL, Seimon TA, Tinkelenberg AH, Wilcox LJ, Lee E et al. Loss of subcellular lipid transport due to ARV1 deficiency disrupts organelle homeostasis and activates the unfolded protein response. J Biol Chem 2011; 286: 11951–11959.

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