Control of mRNA translation preserves endoplasmic reticulum function in beta cells and maintains glucose homeostasis-Reference-Cited by-同舟云学术

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Control of mRNA translation preserves endoplasmic reticulum function in beta cells and maintains glucose homeostasis

Published:2005-06-26 Issue:7 Volume:11 Page:757-764
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Scheuner Donalyn,Mierde Dirk Vander,Song Benbo,Flamez Daisy,Creemers John W M,Tsukamoto Katsura,Ribick Mark,Schuit Frans C,Kaufman Randal J

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1259.pdf

Reference44 articles.

1. Zimmet, P., Alberti, K.G. & Shaw, J. Global and societal implications of the diabetes epidemic. Nature 414, 782–787 (2001).

2. Maechler, P. & Wollheim, C.B. Mitochondrial function in normal and diabetic beta-cells. Nature 414, 807–812 (2001).

3. Hinke, S.A., Hellemans, K. & Schuit, F.C. Plasticity of the beta cell insulin secretory competence: preparing the pancreatic beta cell for the next meal. J. Physiol. (Lond.) 558, 369–380 (2004).

4. Itoh, N. & Okamoto, H. Translational control of proinsulin synthesis by glucose. Nature 283, 100–102 (1980).

5. Schuit, F.C. In't Veld, P.A. & Pipeleers, D.G. Glucose stimulates proinsulin biosynthesis by a dose-dependent recruitment of pancreatic beta cells. Proc. Natl Acad. Sci. USA 85, 3865–3869 (1988).

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