Pediatric non–Down syndrome acute megakaryoblastic leukemia is characterized by distinct genomic subsets with varying outcomes
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng.3772.pdf
Reference41 articles.
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2. Athale, U.H. et al. Biology and outcome of childhood acute megakaryoblastic leukemia: a single institution's experience. Blood 97, 3727–3732 (2001).
3. Barnard, D.R., Alonzo, T.A., Gerbing, R.B., Lange, B. & Woods, W.G. Comparison of childhood myelodysplastic syndrome, AML FAB M6 or M7, CCG 2891: report from the Children's Oncology Group. Pediatr. Blood Cancer 49, 17–22 (2007).
4. Ma, Z. et al. Fusion of two novel genes, RBM15 and MKL1, in the t(1;22)(p13;q13) of acute megakaryoblastic leukemia. Nat. Genet. 28, 220–221 (2001).
5. Gruber, T.A. et al. An Inv(16)(p13.3q24.3)-encoded CBFA2T3-GLIS2 fusion protein defines an aggressive subtype of pediatric acute megakaryoblastic leukemia. Cancer Cell 22, 683–697 (2012).
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