Aicardi–Goutières syndrome and the type I interferonopathies
Author:
Publisher
Springer Science and Business Media LLC
Subject
Energy Engineering and Power Technology,Fuel Technology
Link
http://www.nature.com/articles/nri3850.pdf
Reference106 articles.
1. Gresser, I. et al. Interferon-induced disease in mice and rats. Ann. NY Acad. Sci. 350, 12–20 (1980). A summary of the first experimental data indicating that high levels of IFN might be detrimental in mammals.
2. Aicardi, J. & Goutières, F. A progressive familial encephalopathy in infancy with calcifications of the basal ganglia and chronic cerebrospinal fluid lymphocytosis. Ann. Neurol. 15, 49–54 (1984).
3. Lebon, P. et al. Intrathecal synthesis of interferon-α in infants with progressive familial encephalopathy. J. Neurol. Sci. 84, 201–208 (1988).
4. Crow, Y. J. et al. Cree encephalitis is allelic with Aicardi–Goutières syndrome: implications for the pathogenesis of disorders of interferon α metabolism. J. Med. Genet. 40, 183–187 (2003).
5. Crow, Y. J. et al. Mutations in the gene encoding the 3′–5′ DNA exonuclease TREX1 cause Aicardi–Goutières syndrome at the AGS1 locus. Nat. Genet. 38, 917–920 (2006).
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