Structural basis for MOF and MSL3 recruitment into the dosage compensation complex by MSL1-Reference-Cited by-同舟云学术

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Structural basis for MOF and MSL3 recruitment into the dosage compensation complex by MSL1

Published:2011-01-09 Issue:2 Volume:18 Page:142-149
ISSN:1545-9993
Container-title:Nature Structural & Molecular Biology
language:en
Short-container-title:Nat Struct Mol Biol

Author:

Kadlec Jan,Hallacli Erinc,Lipp Michael,Holz Herbert,Sanchez-Weatherby Juan,Cusack Stephen,Akhtar Asifa

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology,Structural Biology

Link

http://www.nature.com/articles/nsmb.1960.pdf

Reference39 articles.

1. Straub, T. & Becker, P.B. Dosage compensation: the beginning and end of generalization. Nat. Rev. Genet. 8, 47–57 (2007).

2. Heard, E. & Disteche, C.M. Dosage compensation in mammals: fine-tuning the expression of the X chromosome. Genes Dev. 20, 1848–1867 (2006).

3. Mendjan, S. & Akhtar, A. The right dose for every sex. Chromosoma 116, 95–106 (2007).

4. Gelbart, M.E. & Kuroda, M.I. Drosophila dosage compensation: a complex voyage to the X chromosome. Development 136, 1399–1410 (2009).

5. Smith, E.R. et al. A human protein complex homologous to the Drosophila MSL complex is responsible for the majority of histone H4 acetylation at lysine 16. Mol. Cell. Biol. 25, 9175–9188 (2005).

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1. N-Terminus of Drosophila Melanogaster MSL1 Is Critical for Dosage Compensation;2024-09-12

2. N-Terminus of Drosophila Melanogaster MSL1 Is Critical for Dosage Compensation;2024-09-12

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4. KAT8 beyond Acetylation: A Survey of Its Epigenetic Regulation, Genetic Variability, and Implications for Human Health;Genes;2024-05-17

5. N-Terminus of Drosophila Melanogaster MSL1 Is Critical for Dosage Compensation;2024-04-15

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