Thromboxane in the pathogenesis of glomerular injury in diabetes
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference49 articles.
1. Suppression of urinary albumin excretion in diabetic rats by 4′ (imidazol-l-yl) acetophenone, a selective inhibitor of thromboxane synthesis;Craven;J Lab Clin Med,1990
2. Altered steady-state mRNA levels of basement membrane proteins in diabetic mouse kidneys and thromboxane synthetase inhibition;Ledbetter;Diabetes,1990
3. A thromboxane synthetase inhibitor (TSI) ameliorates proteinuria induced by high protein feeding in diabetic rats;Collins;Kidney Int,1990
4. Contribution of platelet thromboxane production to enhanced urinary excretion and glomerular production of thromboxane and to the pathogenesis of albuminuria in the streptozotocin diabetic rat;DeRubertis;Metabolism,1992
5. Effects of a selective thromboxane synthetase inhibitor OKY-046 on experimental diabetic nephropathy;Hora;Nephron,1990
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