Wnt1/βcatenin injury response activates the epicardium and cardiac fibroblasts to promote cardiac repair-Reference-Cited by-同舟云学术

Wnt1/βcatenin injury response activates the epicardium and cardiac fibroblasts to promote cardiac repair

Published:2011-11-15 Issue:2 Volume:31 Page:429-442
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:

Author:

Duan Jinzhu¹²,Gherghe Costin¹²,Liu Dianxin¹²,Hamlett Eric¹²,Srikantha Luxman¹²,Rodgers Laurel¹³,Regan Jenna N¹³,Rojas Mauricio¹,Willis Monte⁴,Leask Andrew⁵,Majesky Mark⁶,Deb Arjun¹²⁷

Affiliation:

1. Department of Medicine, McAllister Heart Institute and Lineberger Comprehensive Cancer Center, University of North Carolina; Chapel Hill NC USA

2. Department of Cell and Molecular Physiology, McAllister Heart Institute and Lineberger Comprehensive Cancer Center, University of North Carolina; Chapel Hill NC USA

3. Department of Genetics, McAllister Heart Institute and Lineberger Comprehensive Cancer Center, University of North Carolina; Chapel Hill NC USA

4. Department of Pathology and Laboratory Medicine, McAllister Heart Institute and Lineberger Comprehensive Cancer Center, University of North Carolina; Chapel Hill NC USA

5. Division of Oral Biology, Schulich School of Medicine and Dentistry, University of Western Ontario; London Ontario Canada

6. Division of Cardiology, Department of Pediatrics, Seattle Children's Research Institute, University of Washington School of Medicine; Seattle WA USA

7. Curriculum in Genetics and Molecular Biology, McAllister Heart Institute and Lineberger Comprehensive Cancer Center, University of North Carolina; Chapel Hill NC USA

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference50 articles.

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5. beta-Catenin stabilization dysregulates mesenchymal cell proliferation, motility, and invasiveness and causes aggressive fibromatosis and hyperplastic cutaneous wounds;Cheon;Proc Natl Acad Sci USA,2002

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