An autism-associated point mutation in the neuroligin cytoplasmic tail selectively impairs AMPA receptor-mediated synaptic transmission in hippocampus-Reference-Cited by-同舟云学术

An autism-associated point mutation in the neuroligin cytoplasmic tail selectively impairs AMPA receptor-mediated synaptic transmission in hippocampus

Published:2011-06-03 Issue:14 Volume:30 Page:2908-2919
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:

Author:

Etherton Mark R¹,Tabuchi Katsuhiko¹²,Sharma Manu¹,Ko Jaewon¹,Südhof Thomas C¹³

Affiliation:

1. Department of Molecular and Cellular Physiology, Stanford University; Palo Alto CA USA

2. Division of Cerebral Structure, Department of Cerebral Research, National Institute for Physiological Sciences; Okazaki Japan

3. Howard Hughes Medical Institute, Stanford University; Palo Alto CA USA

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

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4. Genome-wide analyses of exonic copy number variants in a family-based study point to novel autism susceptibility genes;Bucan;PLoS Genet,2009

5. Neuroligin-3 is a neuronal adhesion protein at GABAergic and glutamatergic synapses;Budreck;Eur J Neurosci,2007

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