Liver-specific deletion of histone deacetylase 3 disrupts metabolic transcriptional networks-Reference-Cited by-同舟云学术

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Liver-specific deletion of histone deacetylase 3 disrupts metabolic transcriptional networks

Published:2008-03-20 Issue:7 Volume:27 Page:1017-1028
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Knutson Sarah K,Chyla Brenda J,Amann Joseph M,Bhaskara Srividya,Huppert Stacey S,Hiebert Scott W

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference67 articles.

1. Translocation breakpoint of acute promyelocytic leukemia lies within the retinoic acid receptor alpha locus.

2. ETO, a Target of t(8;21) in Acute Leukemia, Makes Distinct Contacts with Multiple Histone Deacetylases and Binds mSin3A through Its Oligomerization Domain

3. The p65 (RelA) Subunit of NF-κB Interacts with the Histone Deacetylase (HDAC) Corepressors HDAC1 and HDAC2 To Negatively Regulate Gene Expression

4. The mammalian target of rapamycin regulates lipid metabolism in primary cultures of rat hepatocytes

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