Presynaptic CLC-3 determines quantal size of inhibitory transmission in the hippocampus
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/nn.2775.pdf
Reference28 articles.
1. Stobrawa, S.M. et al. Disruption of ClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus. Neuron 29, 185–196 (2001).
2. Dickerson, L.W. et al. Altered GABAergic function accompanies hippocampal degeneration in mice lacking ClC-3 voltage-gated chloride channels. Brain Res. 958, 227–250 (2002).
3. Yoshikawa, M. et al. CLC-3 deficiency leads to phenotypes similar to human neuronal ceroid lipofuscinosis. Genes Cells 7, 597–605 (2002).
4. Wang, X.Q. et al. CLC-3 channels modulate excitatory synaptic transmission in hippocampal neurons. Neuron 52, 321–333 (2006).
5. Maritzen, T., Keating, D.J., Neagoe, I., Zdebik, A.A. & Jentsch, T.J. Role of the vesicular chloride transporter ClC-3 in neuroendocrine tissue. J. Neurosci. 28, 10587–10598 (2008).
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