GSK-3α/β kinases and amyloid production in vivo-Reference-Cited by-同舟云学术

GSK-3α/β kinases and amyloid production in vivo

Published:2011-12-07 Issue:7376 Volume:480 Page:E4-E5
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Jaworski Tomasz,Dewachter Ilse,Lechat Benoit,Gees Maarten,Kremer Anna,Demedts David,Borghgraef Peter,Devijver Herman,Kügler Seb,Patel Satish,Woodgett Jim R.,Van Leuven Fred

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature10615.pdf

Reference13 articles.

1. Phiel, C. J., Wilson, C. A., Lee, V. M. & Klein, P. S. GSK-3α regulates production of Alzheimer’s disease amyloid-β peptides. Nature 423, 435–439 (2003)

2. Ishiguro, K. et al. Glycogen synthase kinase 3β is identical to tau protein kinase I generating several epitopes of paired helical filaments. FEBS Lett. 325, 167–172 (1993)

3. Spittaels, K. et al. GSK3β phosphorylates protein tau and rescues the axonopathy in the central nervous system of human four-repeat tau transgenic mice. J. Biol. Chem. 275, 41340–41349 (2000)

4. Takashima, A. GSK-3 is essential in the pathogenesis of Alzheimer’s disease. J. Alzheimers Dis. 9 (Suppl.). 309–317 (2006)

5. Terwel, D. et al. Amyloid activates GSK-3β to aggravate neuronal tauopathy in bigenic mice. Am. J. Pathol. 172, 786–798 (2008)

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