The BAH domain of ORC1 links H4K20me2 to DNA replication licensing and Meier–Gorlin syndrome-Reference-Cited by-同舟云学术

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The BAH domain of ORC1 links H4K20me2 to DNA replication licensing and Meier–Gorlin syndrome

Published:2012-03-07 Issue:7392 Volume:484 Page:115-119
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Kuo Alex J.,Song Jikui,Cheung Peggie,Ishibe-Murakami Satoko,Yamazoe Sayumi,Chen James K.,Patel Dinshaw J.,Gozani Or

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature10956.pdf

Reference37 articles.

1. Taverna, S. D., Li, H., Ruthenburg, A. J., Allis, C. D. & Patel, D. J. How chromatin-binding modules interpret histone modifications: lessons from professional pocket pickers. Nature Struct. Mol. Biol. 14, 1025–1040 (2007)

2. Duncker, B. P., Chesnokov, I. N. & McConkey, B. J. The origin recognition complex protein family. Genome Biol. 10, 214 (2009)

3. Bicknell, L. S. et al. Mutations in the pre-replication complex cause Meier-Gorlin syndrome. Nature Genet. 43, 356–359 (2011)

4. Bicknell, L. S. et al. Mutations in ORC1, encoding the largest subunit of the origin recognition complex, cause microcephalic primordial dwarfism resembling Meier-Gorlin syndrome. Nature Genet. 43, 350–355 (2011)

5. Klingseisen, A. & Jackson, A. P. Mechanisms and pathways of growth failure in primordial dwarfism. Genes Dev. 25, 2011–2024 (2011)

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1. Assembly and activation of replicative helicases at origin DNA for replication initiation;Current Opinion in Structural Biology;2024-10

2. Replication licensing regulated by a short linear motif within an intrinsically disordered region of origin recognition complex;Nature Communications;2024-09-13

3. Replication initiation sites and zones in the mammalian genome: Where are they located and how are they defined?;DNA Repair;2024-09

4. Break-induced replication drives large-scale genomic amplifications in cancer cells;2024-08-28

5. Histone variant macroH2A1 regulates synchronous firing of replication origins in the inactive X chromosome;Nucleic Acids Research;2024-08-27

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