Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes-Reference-Cited by-同舟云学术

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Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes

Published:2004-11-21 Issue:12 Volume:6 Page:1221-1228
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Shimizu Shigeomi,Kanaseki Toku,Mizushima Noboru,Mizuta Takeshi,Arakawa-Kobayashi Satoko,Thompson Craig B.,Tsujimoto Yoshihide

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb1192.pdf

Reference18 articles.

1. Baehrecke, E. H. How death shapes life during development. Nature Rev. Mol. Cell Biol. 3, 779–787 (2002).

2. Clarke, P. G. Developmental cell death: morphological diversity and multiple mechanisms. Anat. Embryol. (Berl.) 181, 195–213 (1990).

3. Tsujimoto, Y. Cell death regulation by the Bcl-2 protein family in the mitochondria. J. Cell. Physiol. 195, 158–167 (2003).

4. Lindsten, T. et al. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. Mol. Cell 6, 1389–1399 (2000).

5. Wei, M. C. et al. Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death. Science 292, 727–730 (2001).

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