GSK-3β downregulates Nrf2 in cultured cortical neurons and in a rat model of cerebral ischemia-reperfusion-Reference-Cited by-同舟云学术

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GSK-3β downregulates Nrf2 in cultured cortical neurons and in a rat model of cerebral ischemia-reperfusion

Published:2016-02-03 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Chen Xi,Liu Yuanling,Zhu Jin,Lei Shipeng,Dong Yuan,Li Lingyu,Jiang Beibei,Tan Li,Wu Jingxian,Yu Shanshan,Zhao Yong

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep20196.pdf

Reference36 articles.

1. Doyle, K. P. & Simon, R. P. Mechanisms of ischemic brain damage. Neuropharmacology. 55, 310–318 (2008).

2. Danilov, C. A. et al. Sulforaphane protects astrocytes against oxidative stress and delayed death caused by oxygen and glucose deprivation. Glia. 57, 645–656 (2009).

3. Gu, J. et al. Icariside II enhances Nrf2 nuclear translocation to upregulate phase II detoxifying enzyme expression coupled with the ERK, Akt and JNK signaling path-ways. Molecules. 16, 9234–9244 (2011).

4. Hwang, Y. P. & Jeong, H. G. Ginsenoside Rb1 protects against 6-hydroxydopamine-induced oxidative stress by increasing heme oxygenase-1 expression through an estrogen receptor-related PI3K/Akt/Nrf2-dependent pathway in human dopaminergic cells. Toxicol. Appl. Pharmacol. 242(1), 18–28 (2010).

5. Calkins, M. J. et al. The Nrf2/ARE pathway as a potential therapeutic target in neurodegener-ative disease. Antioxid. Redox. Signal. 11, 497–508 (2009).

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