HCN channels are a novel therapeutic target for cognitive dysfunction in Neurofibromatosis type 1-Reference-Cited by-同舟云学术

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HCN channels are a novel therapeutic target for cognitive dysfunction in Neurofibromatosis type 1

Published:2015-04-28 Issue:11 Volume:20 Page:1311-1321
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Omrani A,van der Vaart T,Mientjes E,van Woerden G M,Hojjati M R,Li K W,Gutmann D H,Levelt C N,Smit A B,Silva A J,Kushner S A,Elgersma Y

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/mp201548.pdf

Reference54 articles.

1. Krab LC, Aarsen FK, de Goede-Bolder A, Catsman-Berrevoets CE, Arts WF, Moll HA et al. Impact of neurofibromatosis type 1 on school performance. J Child Neurol 2008; 23: 1002–1010.

2. Hyman SL, Shores A, North KN . The nature and frequency of cognitive deficits in children with neurofibromatosis type 1. Neurology 2005; 65: 1037–1044.

3. Costa RM, Federov NB, Kogan JH, Murphy GG, Stern J, Ohno M et al. Mechanism for the learning deficits in a mouse model of neurofibromatosis type 1. Nature 2002; 415: 526–530.

4. Silva AJ, Frankland PW, Marowitz Z, Friedman E, Laszlo GS, Cioffi D et al. A mouse model for the learning and memory deficits associated with neurofibromatosis type I. Nat Genet 1997; 15: 281–284.

5. van der Vaart T, van Woerden GM, Elgersma Y, de Zeeuw CI, Schonewille M . Motor deficits in neurofibromatosis type 1 mice: the role of the cerebellum. Genes Brain Behav 2011; 10: 404–409.

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