S6K1 determines the metabolic requirements for BCR-ABL survival-Reference-Cited by-同舟云学术

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S6K1 determines the metabolic requirements for BCR-ABL survival

Published:2012-03-05 Issue:4 Volume:32 Page:453-461
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Barger J F,Gallo C A,Tandon P,Liu H,Sullivan A,Grimes H L,Plas D R

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc201270.pdf

Reference50 articles.

1. Hsu PP, Sabatini DM . Cancer cell metabolism: warburg and beyond. Cell 2008; 134: 703–707.

2. Plas DR, Talapatra S, Edinger AL, Rathmell JC, Thompson CB . Akt and Bcl-xL promote growth factor-independent survival through distinct effects on mitochondrial physiology. J Biol Chem 2001; 276: 12041–12048.

3. Rathmell JC, Fox CJ, Plas DR, Hammerman PS, Cinalli RM, Thompson CB . Akt-directed glucose metabolism can prevent Bax conformation change and promote growth factor-independent survival. Mol Cell Biol 2003; 23: 7315–7328.

4. Buzzai M, Bauer DE, Jones RG, Deberardinis RJ, Hatzivassiliou G, Elstrom RL et al. The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid beta-oxidation. Oncogene 2005; 24: 4165–4173.

5. Schafer ZT, Grassian AR, Song L, Jiang Z, Gerhart-Hines Z, Irie HY et al. Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment. Nature 2009; 461: 109–113.

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