Regulation of autophagy during ECM detachment is linked to a selective inhibition of mTORC1 by PERK-Reference-Cited by-同舟云学术

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Regulation of autophagy during ECM detachment is linked to a selective inhibition of mTORC1 by PERK

Published:2012-11-19 Issue:41 Volume:32 Page:4932-4940
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Avivar-Valderas A,Bobrovnikova-Marjon E,Alan Diehl J,Bardeesy N,Debnath J,Aguirre-Ghiso J A

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2012512.pdf

Reference39 articles.

1. Nelson CM, Bissell MJ . Of extracellular matrix, scaffolds, and signaling: tissue architecture regulates development, homeostasis, and cancer. Annu Rev Cell Dev Biol 2006; 22: 287–309.

2. Schafer ZT, Grassian AR, Song L, Jiang Z, Gerhart-Hines Z, Irie HY et al. Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment. Nature 2009; 461: 109–113.

3. Ng TL, Leprivier G, Robertson MD, Chow C, Martin MJ, Laderoute KR et al. The AMPK stress response pathway mediates anoikis resistance through inhibition of mTOR and suppression of protein synthesis. Cell Death Differ 2012; 19: 501–510.

4. Laplante M, Sabatini DM . mTOR signaling at a glance. J Cell Sci 2009; 122 (Part 20): 3589–3594.

5. Raven JF, Koromilas AE . PERK and PKR: old kinases learn new tricks. Cell Cycle 2008; 7: 1146–1150.

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