Loss of IL36RN Function Does Not Confer Susceptibility to Psoriasis Vulgaris-Reference-Cited by-同舟云学术

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Loss of IL36RN Function Does Not Confer Susceptibility to Psoriasis Vulgaris

Published:2014-01 Issue:1 Volume:134 Page:271-273
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Berki Dorottya M.,Mahil Satveer K.,David Burden A.,Trembath Richard C.,Smith Catherine H.,Capon Francesca,Barker Jonathan N.

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference12 articles.

1. A method and server for predicting damaging missense mutations;Adzhubei;Nat Methods,2010

2. IL-1RL2 and its ligands contribute to the cytokine network in psoriasis;Blumberg;J Immunol,2010

3. Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation;Blumberg;J Exp Med,2007

4. Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesis;Carrier;J Invest Dermatol,2011

5. IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression;Johnston;J Immunol,2011

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4. The role of the interleukin-36 axis in generalized pustular psoriasis: a review of the mechanism of action of spesolimab;Frontiers in Immunology;2023-11-21

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