The molecular basis of CaMKII function in synaptic and behavioural memory
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/nrn753.pdf
Reference126 articles.
1. Soderling, T. R., Chang, B. & Brickey, D. Cellular signaling through multifunctional Ca2+/calmodulin-dependent protein kinase II. J. Biol. Chem. 276, 3719–3722 (2001).
2. Hudmon, A. & Schulman, H. Neuronal Ca2+/calmodulin-dependent protein kinase II: the role of structure and autoregulation in cellular function. Annu. Rev. Biochem. 2002 (10.1146/annurev.biochem.71.110601.135410).
3. Lisman, J. The CaM kinase II hypothesis for the storage of synaptic memory. Trends Neurosci. 17, 406–412 (1994).
4. Bliss, T. V. & Collingridge, G. L. A synaptic model of memory: long-term potentiation in the hippocampus. Nature 361, 31–39 (1993).
5. Malenka, R. C. & Nicoll, R. A. Long-term potentiation — a decade of progress? Science 285, 1870–1874 (1999).Summarizes extensive work that points to a postsynaptic site for the expression of LTP in the CA1 pyramidal cells of the hippocampus.
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