Bmi1 regulates mitochondrial function and the DNA damage response pathway-Reference-Cited by-同舟云学术

Bmi1 regulates mitochondrial function and the DNA damage response pathway

Published:2009-04-29 Issue:7245 Volume:459 Page:387-392
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Liu Jie,Cao Liu,Chen Jichun,Song Shiwei,Lee In Hye,Quijano Celia,Liu Hongjun,Keyvanfar Keyvan,Chen Haoqian,Cao Long-Yue,Ahn Bong-Hyun,Kumar Neil G.,Rovira Ilsa I.,Xu Xiao-Ling,van Lohuizen Maarten,Motoyama Noboru,Deng Chu-Xia,Finkel Toren

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature08040.pdf

Reference38 articles.

1. Park, I. K. et al. Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells. Nature 423, 302–305 (2003)

2. Molofsky, A. V. et al. Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation. Nature 425, 962–967 (2003)

3. van der Lugt, N. M. et al. Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene. Genes Dev. 8, 757–769 (1994)

4. Jacobs, J. J., Kieboom, K., Marino, S., DePinho, R. A. & van Lohuizen, M. The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus. Nature 397, 164–168 (1999)

5. Bruggeman, S. W. et al. Ink4a and Arf differentially affect cell proliferation and neural stem cell self-renewal in Bmi1-deficient mice. Genes Dev. 19, 1438–1443 (2005)

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