Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation-Reference-Cited by-同舟云学术

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Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation

Published:1997-05 Issue:6632 Volume:387 Page:516-519
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Baskaran R.,Wood L. D.,Whitaker L. L.,Canman C. E.,Morgan S. E.,Xu Y.,Barlow C.,Baltimore D.,Wynshaw-Boris A.,Kastan M. B.,Wang J. Y. J.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/387516a0.pdf

Reference25 articles.

1. Savitsky, K. et al. The complete sequence of the coding region of the ATM gene reveals similarity to cell cycle regulators in different species. Hum. Mol. Genet. 4, 2025–2032 (1995).

2. Savitsky, K. et al. A single ataxia telangiectasia gene with a product similar to PI-3 kinase. Science 268, 1749–1753 (1995).

3. Barlow, C. et al. Atm-deficient mice: a paradigm of ataxia telangiectasia. Cell 86, 159–171 (1996).

4. Xu, Y. et al. Targeted disruption of ATM leads to growth retardation, chromosomal fragmentation during meiosis, immune defects and thymic lymphoma. Genes Dev. 10, 2411–2422 (1996).

5. Xu, Y. & Baltimore, D. Dual roles of ATM in the cellular response to radiation and in cell growth control. Genes Dev. 10, 2401–2410 (1996).

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