TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction-Reference-Cited by-同舟云学术

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TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction

Published:2014-11-26 Issue:4 Volume:8 Page:828-840
ISSN:1933-0219
Container-title:Mucosal Immunology
language:en
Short-container-title:Mucosal Immunol

Author:

Van Hauwermeiren F,Vandenbroucke R E,Grine L,Lodens S,Van Wonterghem E,De Rycke R,De Geest N,Hassan B,Libert C

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/mi2014112.pdf

Reference63 articles.

1. Tracey, K.J. et al. Anti-cachectin/TNF monoclonal antibodies prevent septic shock during lethal bacteraemia. Nature 330, 662–664 (1987).

2. Feldmann, M. & Maini, R.N. Lasker Clinical Medical Research Award. TNF defined as a therapeutic target for rheumatoid arthritis and other autoimmune diseases. Nat. Med. 9, 1245–1250 (2003).

3. van Dullemen, H.M. et al. Treatment of Crohn’s disease with anti-tumor necrosis factor chimeric monoclonal antibody (cA2). Gastroenterology 109, 129–135 (1995).

4. Huys, L. et al. Type I interferon drives tumor necrosis factor-induced lethal shock. J. Exp. Med. 206, 1873–1882 (2009).

5. Takahashi, N. et al. IL-17 produced by Paneth cells drives TNF-induced shock. J. Exp. Med. 205, 1755–1761 (2008).

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